Often, when we speak of a broken heart or a broken heart we are referring to a psychological condition, which arises as a result of emotionally trying events and because of which we feel downhill.
However, around the early 1990s, Japanese researchers demonstrated the existence of cardiomyopathy - a myocardial disease, the heart muscle - that arises precisely as a result of severe emotional stress (grief, severe affliction, great anger, etc.). ) or physical stress (exaggerated exertion, delicate surgery, etc.).
This problem, for which the term broken heart syndrome or takotsubo cardiomyopathy has been coined, is generally temporary, but it can also lead to the death of its victim.
SOME POSSIBLE CAUSES
As in all cardiomyopathies, takotsubo cardiomyopathy is also characterized by an anatomical modification of the myocardium which negatively affects its functional level, reducing the contractile capacity of the heart.
But what exactly does this induce?
Currently, the exact pathological mechanism is unclear and there are several theories.
According to some scholars of the disease, strong emotional or physical stresses would induce a strong release of hormones (probably adrenaline and noradrenaline ), which are harmful to myocardial cells. In fact, it would seem that, after their release, the hormones go to "stun" the muscular tissue that constitutes the left ventricle, alternating first the form and then the function.
